Understanding Atrial Fibrillation Mechanisms

Sep 12, 2024

Atrial Fibrillation (AF)

Key Definitions

  • Atrial Fibrillation (AF): Most common arrhythmia; characterized by an irregularly irregular ventricular rhythm and absence of discrete P waves.
  • Supraventricular Tachycardia: AF is classified as this type of arrhythmia.

Mechanism of AF

  • Atrial Activity: In AF, the atria quiver instead of contracting synchronously, leading to irregular and rapid beats.
  • Clot Formation: Most concerning complication is clot formation due to stasis, often in the left atrium, which can lead to embolism and stroke.

Heart Conduction System

  • Sinoatrial Node: The dominant pacemaker; sends impulses for synchronized atrial contraction (P wave on ECG).
  • Atrioventricular Node: Conducts impulses slower; acts as a gatekeeper to the ventricles.
  • Bundle of His and Purkinje Fibers: Impulses travel rapidly here, leading to ventricular contraction (QRS complex on ECG).

Triggers of AF

  • Ectopic Foci: Pacemaker cells located abnormally; commonly found in the left atrium near pulmonary veins.
  • Re-entry Circuit: Can result from ischemic heart disease, age, hypertension, leading to structural changes in atrial myocardium.

Types of Atrial Fibrillation

  • Paroxysmal AF: Lasts less than 7 days; often resolves spontaneously.
  • Persistent AF: Lasts longer than 7 days; may require intervention.
  • Permanent AF: Restoration to sinus rhythm is impossible; progressive remodeling occurs.

ECG Findings

  • Classic Characteristics: Absent P waves, irregularly irregular ventricular rate, F waves (fibrillatory waves), and varying heart rates (100-280 bpm).

Diagnosis

  • Paroxysmal AF: Diagnosed via Holter monitor or implantable loop recorder.
  • Persistent AF: Diagnosed with ECG.

Management Strategies

Acute Management

  • Unstable Patients: Require synchronized electrical cardioversion; followed by amiodarone infusion.
  • Stable Patients: Can use chemical cardioversion with antiarrhythmics (e.g., flecainide, sotalol).
  • Anticoagulation: Important if AF has been present for over 48 hours to rule out thrombus formation; transesophageal echocardiogram or four weeks of anticoagulation may be necessary prior to cardioversion.

Long-term Management

  • Rhythm Control vs. Rate Control: Depends on symptomatology, side effects, and contraindications.
  • Rate Control Medications: Beta blockers, calcium channel blockers, digoxin.
  • Rhythm Control Medications: Sotalol, amiodarone.
  • Anticoagulation: Assess using CHADS-VASc score to determine stroke risk; different anticoagulants for valvular vs. non-valvular AF.
    • Valvular AF: Warfarin is mainstay.
    • Non-Valvular AF: NOACs (e.g., rivaroxaban, apixaban) are preferred.

Catheter Ablation

  • Effective for paroxysmal AF; targets ectopic foci in pulmonary veins. More complex procedures needed for advanced AF.