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Citric Acid Cycle and its Regulation
May 20, 2024
Lecture Notes: Citric Acid Cycle Regulation
Overview of the Citric Acid Cycle (CAC)
Begins with:
Acetyl CoA combining with oxaloacetate to form citrate.
Acetyl CoA:
Two carbon molecule.
Key Outputs:
Acetyl CoA is oxidized and exits as CO2; production of NADH, FADH2, and GTP.
Overall Chemical Reaction of CAC
Reactants:
Acetyl CoA, NAD+, FAD, GDP.
Products:
CO2, NADH, FADH2, GTP.
Importance:
NADH and FADH2 go to the electron transport chain for ATP production.
Difference from Glycolysis
Glycolysis:
Can be turned on/off based on glucose availability.
CAC:
Always on but at varying speeds to meet energy needs of the cell.
Modes of CAC Regulation
Allosteric Regulation
Definition:
Molecules bind to enzymes at non-active sites causing conformational changes.
Types:
Allosteric activators or inhibitors based on the effect on enzyme activity.
Response:
Adjusts to the energy needs of the cell.
Substrate Availability
High Acetyl CoA:
Fast cycle; low Acetyl CoA: Slow cycle.
**Examples: **
Citrate in high ATP conditions shuttles acetyl CoA out for fatty acid synthesis, slowing CAC.
Amino acids convert into CAC intermediates during starvation.
Key Irreversible Reactions in CAC
Characteristics:
Large negative ΔG, good targets for regulation.
Key Reactions:
Oxaloacetate + Acetyl CoA → Citrate (Citrate Synthase)
Isocitrate → α-ketoglutarate (Isocitrate Dehydrogenase)
α-ketoglutarate → Succinyl CoA (α-Ketoglutarate Dehydrogenase)
Allosteric Regulators
Inhibitors
NADH:
Product of CAC; signals to slow down CAC as it accumulates.
Inhibits: Citrate Synthase, Isocitrate Dehydrogenase, α-Ketoglutarate Dehydrogenase.
ATP:
High ATP signals sufficient energy, slows CAC.
Inhibits: Citrate Synthase, Isocitrate Dehydrogenase.
Products:
Feedback inhibition.
Citrate: Inhibits Citrate Synthase.
Succinyl CoA: Inhibits Citrate Synthase, α-Ketoglutarate Dehydrogenase.
Activators
ADP:
Signals low ATP, activates CAC to produce more ATP.
Activates: Citrate Synthase, Isocitrate Dehydrogenase.
Calcium (Ca2+):
Influx during muscle contraction signals high energy need.
Activates: Isocitrate Dehydrogenase, α-Ketoglutarate Dehydrogenase.
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