🧬

Neural Tube Defects and Folate

Jul 4, 2025

Overview

This lecture discusses the genetic and environmental causes of neural tube defects (NTDs), focusing on the role of folate metabolism, epigenetic regulation, and epidemiologic evidence for folate dose-dependency in NTD prevention.

Causes of Neural Tube Defects (NTDs)

  • NTDs have both genetic and environmental (epigenetic) risk factors.
  • Genetic mutations and environmental influences impact neural tube closure during embryonic development.
  • Many NTDs involve defects in the folate metabolic pathway.

Folate Pathway and Its Importance

  • Folate is essential for the synthesis of purines and pyrimidines, the building blocks of nucleotides.
  • Folate provides methyl groups needed for methylation of DNA and other macromolecules.
  • DNA methylation, influenced by folate, regulates gene expression critical to neural tube formation.

Molecular Pathways Linked to NTDs

  • The planar cell polarity pathway (studied in mice) is crucial for proper folic acid metabolism and neural tube closure.
  • Transmembrane and cytosolic proteins facilitate normal folic acid metabolism in embryonic cells.
  • Failure of neuroectoderm and cutaneous ectoderm dissociation leads to NTDs, usually around day 25–26 of embryonic development.

Epidemiological Evidence: Folate and NTD Risk

  • Studies show a dose-dependent relationship between maternal red blood cell folate levels and NTD incidence.
  • Higher maternal folate levels correspond to lower rates of NTDs in offspring.
  • Krista Kreider (CDC) conducted large population studies in Asia demonstrating this linear relationship.

Key Terms & Definitions

  • Neural Tube Defect (NTD) — Birth defect arising from incomplete closure of the neural tube during embryogenesis.
  • Folate — A B-vitamin (B9) vital for DNA synthesis and methylation processes.
  • Methylation — Addition of a methyl group to DNA or other molecules, affecting gene regulation.
  • Planar Cell Polarity Pathway — A molecular signaling pathway involved in coordinating cell orientation and folic acid metabolism.
  • Dose Dependency — The relationship where the effect changes as the dose of a substance (e.g., folate) increases or decreases.

Action Items / Next Steps

  • Review embryology of neural tube formation and failure points.
  • Study the folate metabolic pathway and its role in DNA synthesis.
  • Read Krista Kreider’s epidemiologic paper on maternal folate and NTD risk.