Development of the Placenta
Overview
- Discuss development of the placenta
- Hormonal functions of the placenta
- Clinical correlations
- Reference video on cleavage and implantation
Implantation and Early Development
- Fertilization: Sperm + secondary oocyte (metaphase 2) = zygote
- Cleavage Stages: Zygote divides --> 2-cell stage --> 4-cell stage --> 8-cell stage --> 16-cell (morula)
- Morula: Hollow ball of 16+ cells with a zona pellucida (ZP)
- Blastocyst Formation: Water enters morula, forming a blastocoel --> Differentiates into outer cell mass (trophoblast) and inner cell mass (embryoblast)
- Implantation: Blastocyst attaches to the endometrium, which later differentiates into decidua (decidua basalis and decidua functionalis)
Detailed Implantation Process
- Attachment: Loose connection
- Trophoblast microvilli adhere to endometrial pinopods
- Stronger attachment: Integrins on trophoblast bind to selectins and collagen on endometrium
- Chemokines stabilize adhesion
- Differentiation of Trophoblasts: CytoTrophoblast & SynCytioTrophoblast
- CytoTrophoblast: Proliferates and contributes to syncytiotrophoblast
- SynCytioTrophoblast: Releases hydrolytic enzymes and HCG, ensuring endometrial integrity
- Syncytiotrophoblast Invasion: Releases enzymes to invade stromal tissue
- HCG Secretion: Maintains progesterone levels to prevent menstruation until placenta takes over
Villous Development
- Primary Villi: Cytotrophoblast penetrates syncytiotrophoblast, forming outer cytotrophoblastic shell
- Secondary Villi: Extraembryonic mesoderm invades primary villi
- Tertiary Villi: Mesoderm forms blood vessels, allowing for nutrient and gas exchange
Formation of Chorionic Structures
- Extraembryonic Coelom: Forms around Day 12, becoming chorionic cavity
- Chorionic Plate: Develops from extraembryonic mesoderm
- Outer Cytotrophoblastic Shell: Formed by cytotrophoblast penetrating syncytiotrophoblast
- Formation of Connecting Stalk: Converts into the umbilical cord with arteries and veins
Maternal-Fetal Exchange and Circulation
- Blood Flow Route: Umbilical arteries > Chorionic arteries > Cotyledon arteries > Capillaries in villi
- Gas and Nutrient Exchange: Takes place between intervillous spaces filled with maternal blood and fetal blood vessels
Placental Development Timeline
- Weeks 4-8: Tertiary villi become more branched for increased surface area
- 4th-5th Month: Cotyledon formation due to septations of decidua
- >20 Weeks: Cytotrophoblastic layer regresses, enhancing exchange efficiency
Placental Linings and Associated Structures
- Layers: Amniotic membrane, chorionic membrane, decidua capsularis, decidua parietalis
- Placental Formation: Chorionic frondosum (fetal component) interacts with decidua basalis (maternal component)
- Fusion: Decidua capsularis and decidua parietalis fuse, obliterating the uterine cavity
Hormonal Functions of the Placenta
- Estrogen & Progesterone: Maintain endometrial lining, increase secretions, and support fetal development
- Human Chorionic Gonadotropin (HCG): Maintains corpus luteum function
- Thyroid Hormone: Supports CNS development
- Human Placental Lactogen: Modifies maternal metabolism to support fetal needs (increases glucose and fatty acid availability)
- Relaxin: Relaxes pubic symphysis ligaments
- Corticotropin-Releasing Hormone (CRH): Promotes cortisol production for lung development and surfactant production
Metabolic and Exchange Functions
- Gas Exchange: Oxygen to fetus, CO2 removal
- Nutrient Delivery: Glucose, amino acids, fatty acids, water-soluble vitamins, IgG antibodies (passive immunity)
- Waste Removal: Urea, uric acid
Pathogenic Concerns (TORCH Infections)
- Toxoplasmosis: Toxoplasma gondii
- Others: HIV, syphilis, hepatitis B
- Rubella: Can cause cardiac and congenital defects
- CMV (Cytomegalovirus)
- Herpes Simplex Virus: Type 2
- Zika Virus: Vertical transmission risk
Clinical Correlations
- Placenta Accreta, Increta, Percreta: Abnormal placenta attachment leading to complications
- Rhesus (Rh) Incompatibility: Condition where Rh-negative mother has immune response against Rh-positive fetal blood cells
Treatment: RhoGAM to prevent hemolytic disease