Lecture on Fatty Liver Disease

Understanding Fatty Liver

Fatty liver diagnosis often involves liver function tests (e.g., A or ALT), ultrasounds, MRIs, or CAT scans.
Common explanations from mainstream medicine include unknown viruses or genetic factors.
Traditional management includes monitoring, eventual discussion about liver transplants.

Cirrhosis: Liver becomes dysfunctional, abdomen fills with fluid (ascites), requiring medical interventions (e.g., shunt).
Liver Transplant: Involves significant risks, postoperative complications, reliance on immunosuppressive drugs.

Many mainstream and alternative treatments (e.g., milk thistle) are criticized as ineffective.
True cause often linked to bowel flora issues like small intestinal fungal overgrowth.

Hepatic Denovo Lipogenesis
- Liver converts carbohydrates (especially amalopectin A from wheat/grains) and sugars (glucose, fructose) into triglycerides.
- Triglycerides accumulate in the liver as fat.
- Misconception: Dietary fat is not the cause.
Insulin Resistance
- Organs don't respond properly to insulin, leading to overproduction by the pancreas.
- Exacerbates liver's conversion of carbohydrates to triglycerides.
Endotoxemia
- Fecal microbes release toxins (lipopolysaccharides) into the bloodstream.
- Toxins reach the liver via the portal vein, worsening triglyceride accumulation.

Eliminate wheat, grains, and sugars to stop the liver from producing excess triglycerides.
Do not restrict fats; focus on reducing carbohydrate intake.

Restore levels of magnesium, iodine, omega-3 fatty acids, and vitamin D to reduce insulin resistance.

Use a combination of SIBO Yogurt containing:
- Lactobacillus reuteri
- Lactobacillus gasseri
- Bacillus subtilis
These microbes help colonize the small intestine and produce natural antibiotics (bacteriocins) to kill harmful bacteria.