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Obeticholic Acid (OCA) Lecture Notes
Jun 23, 2024
Lecture Notes: Obeticholic Acid (OCA)
Introduction
Drug:
Obeticholic acid (OCA)
Class:
Farnesoid X receptor agonist
Function:
Reduces bile acid levels in hepatocytes
Derived from:
Chenodeoxycolic acid (secondary bile acid)
Mechanism of Action
Bile Acids and Receptors
Acts on farnesoid X receptors (FXR)
Reduces hepatic apoptosis and cirrhosis
Primary Biliary Cholangitis (PBC)
Condition involving increased gallstones and decreased biliary flow leading to cholestasis and inflammation
OCA reduces bile acid levels, used in PBC treatment
Usage
Combination with UDCA:
Ursodeoxycholic acid (often used for PBC)
Patient Sensitivity:
Should be given to patients who do not tolerate UDCA
Hepatic Decompensation:
OCA should be restricted to avoid severe hepatic effects
Chemical Nature
Structure:
Cholan-24-oic acid; 3,7-Dihydroxy; 6 alpha-ethyl
Synthesis Pathway:
Cholesterol → Bile acids (mediated by CYP7A1)
Transporters Affected
NTCP:
Sodium taurocholate co-transporting polypeptide (uptake of bile acids)
BSEP:
Bile salt export pump (secretion of bile acids)
ASBT:
Apical sodium bile acid transporter (absorption of bile acids)
Effects:
Inhibits NTCP (reduces bile acid uptake)
Promotes BSEP (increases bile acid secretion)
Inhibits ASBT (reduces bile acid absorption)
Induces FGF19 (fibroblast growth factor 19), further reducing bile acid synthesis
Precautions
Hepatic Issues:
Can cause hepatic decompensation
Symptoms to Watch:
Jaundice, ascites, GI bleeding
Pruritus:
Severe itching causing sleep disturbance
HDL Cholesterol:
Can decrease HDL levels, increasing cardiovascular risks
Side Effects
Common Side Effects:
Abdominal pain, fatigue, arthralgia (joint pain)
Pruritus (itching), dizziness, constipation
Eczema, peripheral edema, palpitations, pyrexia (fever)
Dosage
Form:
Tablets (5 mg and 10 mg)
Initial Dose:
5 mg once daily
Increased Dose:
Can be increased to 10 mg once daily after 3 months
Hepatic Dysfunction:
Reduce to 5 mg once weekly
Summary
Effectiveness:
Reduces hepatic apoptosis and biliary cirrhosis by affecting bile acid levels and transporters
Combination with UDCA:
Enhances efficacy of PBC treatment
Careful Use:
Needed in patients with hepatic decompensation
Conclusion
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