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Krebs Cycle Summary

Jun 14, 2025

Overview

This lecture covers the Krebs cycle (also known as the citric acid or tricarboxylic acid cycle), including its intermediates, key enzymes, regulation, and products.

Krebs Cycle Overview

  • The Krebs cycle occurs in the mitochondria and is central to cellular aerobic respiration.
  • Pyruvate from glycolysis is converted to acetyl-CoA, which combines with oxaloacetate to form citrate.
  • One cycle processes one acetyl-CoA; since one glucose makes two pyruvates, the cycle runs twice per glucose.

Key Intermediates & Mnemonic

  • Intermediates in order: Citrate, Isocitrate, α-Ketoglutarate, Succinyl-CoA, Succinate, Fumarate, Malate, Oxaloacetate.
  • Mnemonic: “Citrate Is Krebs’ Starting Substrate For Making Oxaloacetate.”

Major Enzymes & Steps

  • Citrate synthase: Combines acetyl-CoA and oxaloacetate to make citrate; highly regulated, irreversible.
  • Aconitase: Converts citrate to isocitrate; reaction is reversible.
  • Isocitrate dehydrogenase: Converts isocitrate to α-ketoglutarate; produces NADH, releases CO₂, irreversible.
  • α-Ketoglutarate dehydrogenase: Converts α-ketoglutarate to succinyl-CoA; produces NADH, releases CO₂, irreversible.
  • Succinyl-CoA synthetase: Converts succinyl-CoA to succinate; produces GTP (then ATP by substrate-level phosphorylation).
  • Succinate dehydrogenase (Complex II): Converts succinate to fumarate; produces FADH₂; part of the electron transport chain.
  • Fumarase: Converts fumarate to malate.
  • Malate dehydrogenase: Converts malate to oxaloacetate; produces NADH.

Regulation of the Cycle

  • Citrate synthase: Inhibited by ATP, NADH, citrate, and succinyl-CoA; stimulated by ADP.
  • Isocitrate dehydrogenase: Inhibited by ATP; stimulated by ADP and Ca²⁺.
  • α-Ketoglutarate dehydrogenase: Inhibited by NADH and succinyl-CoA; stimulated by Ca²⁺.

Cycle Products (per glucose, i.e., 2 cycles)

  • 6 NADH, 2 FADH₂, 2 ATP (by substrate-level phosphorylation), 4 CO₂.

Clinical Connections

  • Mutations or inhibitors (e.g., fluoroacetate, mutations in succinate dehydrogenase or fumarase) can cause cell dysfunction or tumors (gliomas, pheochromocytoma, leiomyomas).
  • α-Ketoglutarate is a cofactor for histone demethylase, influencing gene expression; disruptions can lead to cancer.

Key Terms & Definitions

  • Krebs cycle (Citric acid cycle) — Sequence of reactions that oxidize acetyl-CoA to CO₂ and generate NADH, FADH₂, and ATP.
  • Substrate-level phosphorylation — Direct ATP (or GTP) formation from ADP (or GDP) using energy from a substrate.
  • Dehydrogenase — Enzyme catalyzing redox reactions, often producing NADH or FADH₂.
  • Decarboxylation — Removal of CO₂ from a molecule.
  • Aconitase — Enzyme isomerizing citrate to isocitrate.
  • Succinate dehydrogenase — Enzyme functioning in both the Krebs cycle and electron transport chain (Complex II).

Action Items / Next Steps

  • Memorize the order of intermediates using the provided mnemonic.
  • Review and understand regulation of key enzymes (citrate synthase, isocitrate dehydrogenase, α-ketoglutarate dehydrogenase).
  • Practice drawing the cycle and noting where products are formed.