Transcript for:
L19_Introduction to Epidemiology and Population Sciences

Title: URL Source: blob://pdf/d2b11ed5-931b-4767-9937-9220a0d7c1af Markdown Content: Introduction to Epidemiology and Population Sciences Prof Mieke Van Hemelrijck Transforming cancer OUtcomes through Research (TOUR) Kings College London Overview TOUR team Modules Introduction to Epidemiology and Population Sciences Basic Vocabulary and Concepts Details of the Modules Learning outcomes After this lecture you should be able to: Define the term epidemiology and explain its relevance in the context of population health. Understand the scope of epidemiology, including key concepts like exposure, outcome, and risk. Differentiate between prevalence and incidence, and describe how these measures are used in public health. Describe the concept of risk and distinguish between relative and absolute measures of effect. Identify common sources of confounding and explain how confounding can distort associations in epidemiological research. Understand strategies to control for confounding at the design and analysis stages of studies. Define effect modification and distinguish it from confounding using relevant examples. Appreciate the historical context of epidemiology, including the significance of Dr John Snows work in London. Recognise the role of epidemiology and population sciences in medical fields such as cancer research and healthcare outcomes. TOUR To use science to improve healthcare outcomes by translating oncology research into clinical practice. A heterogeneous set of activities with clinical cancer epidemiology at the core. Provide a methodology for researching prevention, early diagnosis and detection, treatment outcomes, and living with and beyond cancer. Role of clinical epidemiology across the entire cancer patient pathway. Modules 4BBY1002: Introduction to Clinical Exercise Epidemiology 5MCS1000: Introduction to Epidemiology & Population Sciences 15 credits in Semester B 6MCS1001: Advanced Epidemiology & Population Sciences 15 credits in Semester B 6MCS1000: Cancer in Context: Public Health & Clinical Epidemiology 15 Credits in Semester A Introduction # to # Epidemiolog # yIt all started in London ... with a ## pump handle > 7 Dr John Snow (1813-1858) The John Snow Pub Broadwick Street, Soho What is epidemiology ? According to the world health organization: Epidemiology is the study of the distribution and determinants of health-related states or events (including disease), and the application of this study to the control of diseases and other health problems > 8 # What is epidemiology? The study of the distribution and determinants of disease in occurrence in human populations Outcome of interest Exposure of interest Biostatistical methods > 9 ## What is epidemiology? > 10 Ultimate aim: To prevent disease by eliminating, reducing exposure to its determinants Method: Identification of disease determinants Context: Medicine, public health The scope of # epidemiology > 11 Exposure Risk factor Cause Outcome Event Effect The scope of # epidemiology > 12 Exposure Risk factor Cause (e) Disease Event Effect (e) Outcome Death Effect (p) Intervention as a consequence of event Cause (p) > Etiognosis (e) Diagnosis Prognosis (p) ## Vocabulary > 13 Study base Exposure Outcome Prevalenc e Incidence Risk Confound er Effect Modifier Study # base > 14 Reference population Source of the study population Population giving rise to the cases Defined before cases appear by a geographical area or some other entity like a cohort study Study base? > 15 ## Exposure An exposure, risk factor, or other characteristic being observed or measured that is hypothesized to influence an event or manifestation > 16 ## Exposure ## ? > 17 ## Outcome > 18 Disease Disease progression Death Comorbidity Questionnaire data Biological endpoints expression levels Outcome ## ? > 19 # Prevalence Proportion of a population found to have a condition at a specific point in time > 20 IARC, Globocan Incidence > 21 IARC, Globocan Proportion of a population found to have developed a condition during a specific period of time. Risk Probability of disease developing in an individual in a specified time interval > 22 ## Risk? > 23 ## Measurements of effect Relative: exposed versus unexposed Absolute: i.e. incidence, prevalence Courtesy of Prof David Spiegelhalter 100 people eating a bacon sandwich every other day ## What does this ## mean? Courtesy of Prof David Spiegelhalter 100 people NOT eating a bacon sandwich every other day ## What does this ## mean? 25g processed meat a day associated with 19% increased risk of getting bowel cancer Such Relative risks are known to exaggerate apparent effect Need absolute risks Around 6% of people will get bowel cancer anyway So what is an 19% increase over 6%? 1.19 times 0.06 = 0.07 > Courtesy of Prof David Spiegelhalter ## Confounding The apparent effect of the exposure of interest is distorted because the effect of an extraneous factor (confounder) is mistaken for or mixed with the actual exposure effect (which may be null), leading to bias. Confounding may be considered a confusion of effects Classic ## example of ## confoundin ## g Increasing trend for prevalence of Down syndrome with increasing birth order = effect of birth order on the occurrence of Down syndrome Effect of birth order is blend of effect of birth order itself and effect of other variable closely correlated with birth order Maternal age is much stronger associated with Down syndrome Mixing effects Another ## example In men baldness is associated with the risk of myocardial infarction Age increases both the likelihood of baldness and the risk of myocardial infarction; thus age confounds the association between baldness and myocardial infarction Criteria ## for a ## confoundi ## ng factor A confounding factor must be a risk factor for the disease A confounding factor must be associated with the exposure under study in the source population A confounding factor must not be affected by the exposure or the disease. Cannot be an intermediate factor. Do the ## criteria ## hold? causes causes Sodium elevated blood heart disease intake pressure Is elevated blood pressure a confounder? # Do the criteria hold? Control of ## confoundi ## ng Randomisation Restriction Select subjects with similar values for confounders, e.g., select only old, male Matching Match on confounding variable to force comparison between cases and controls Stratification Modelling Review of confounding 1. Associated with the outcome of interest independent of its relation to the exposure of interest 2. Associated with the exposure of interest in the study base that produced the cases 3. Cannot be an intermediate variable on the causal chain leading from the exposure of interest to the onset of disease Confounding can be altered at the study design stage: Matching Restriction Stratification Randomization The strength of the association between the exposure and the outcome varies by levels of a third variable Effect modifier = Interaction Term > http://essedunet.nsd.uib.no/cms/topics/regression/7/2.html # Effect # Modification Effect modificatio n? Refers to a situation in which two or more risk factors modify one anothers effects on an outcome. The effect of the physical activity categories sedentary or active on the presence or absence of chronic heart disease might differ depending on some third factor, such as sex; physical activity might reduce the risk of disease among men but not in women. Sex would be the effect modifier. Example ## of effect ## modificati ## on The effect of physical activity assessed by questionnaire on the risk for chronic heart disease (CHD) may depend on BMI. The risk of disease among the least- active people might be greatest among those having the highest BMI. If so, BMI would be considered an effect modifier in the association between activity and CHD. Effect ## modifier vs ## Confoundi ## ng factor Questions? Prof Mieke Van Hemelrijck [email protected] www.kcl.ac.uk/tourteam Details of # Modules on # Epidemiolog # y & # Population # Sciences > 5MCS1000 > 6MCS1000 > 6MCS1001 ## 5MCS1000 L1: Introduction and overview L2: Study Design L3: Bias and Confounding L4: Causality and statistical interpretation L5: Introduction to Randomised Controlled Trials L6: Introduction to Behavioural Sciences L7: Introduction to Implementation Sciences L8: Introduction to Qualitative Research Methods L9: Methods in global cancer: how to utilise mixed methods for comparative systems? L10: Ethics, data integrity, and final summary T1-T6: Review of theory through application of research papers Introduction to Epidemiology & Population Sciences 6MCS1001 L1: Introduction and overview L2: Measurements of life style markers & their correlates L3: Introduction to systematic reviews and meta- analysis L4: Pharmaco-epidemiology L5: Importance of behaviour in health outcomes L6: Real World Evidence L7: Introduction to screening L8: Importance of patient reported outcomes L9: Clinical epidemiology L10: Health systems & services research (cancer) T1-T8: Review of theory through application of research papers Advanced Epidemiology & Population Sciences 6MCS1001 L1: Introduction to clinical cancer epidemiology L2: Introduction to behavioural sciences for cancer interventions L3: Randomised controlled trials and/or/versus Real World Evidence L4: Introduction to mixed methods L5: Quality of Life research for cancer patients L6: The use of Core Outcome Sets in cancer research L7: Linking epidemiology and shared treatment decision-making in the cancer pathway L8: The important of patient and public involvement/engagement L9: Introduction to implementation sciences T1: Case study 1: Global Oncology Bridging research and patient care T2: Case study 2: Health inequalities - Cancer care in the prison population T3: Case study 3: Cancer and Exercise Cancer in Context: Public Health and Clinical Epidemiology