Question 1
Which receptor does histamine attach to on parietal cells?
Question 2
Why is long-term use of H2 blockers and PPIs not recommended?
Question 3
Which receptor does acetylcholine attach to on ECL and parietal cells?
Question 4
Which type of cells in the stomach lining are primarily involved in acid secretion?
Question 5
What happens when proton pumps are shut down by PPIs?
Question 6
How does sodium alginate prevent acid reflux?
Question 7
What is the mechanism of action of H2 blockers?
Question 8
Why are proton pump inhibitors considered stronger than H2 blockers?
Question 9
Which receptor does histamine attach to in the process of acid secretion?
Question 10
How often should sodium alginate be taken for it to be effective?
Question 11
What medication alternative to H2 blockers and PPIs is derived from seaweed?
Question 12
What is the main function of proton pump inhibitors (PPIs)?
Question 13
Which of the following medications shuts down proton pumps?
Question 14
Which neurotransmitter attaches to muscarinic receptors on both ECL and parietal cells?
Question 15
What is a common example of an H2 blocker?
Question 16
What are the two main types of cells in the stomach lining involved in acid secretion?