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Coagulation Cascade Overview

Jul 10, 2025

Overview

This lecture covers the coagulation cascade, outlining its pathways, key steps, end products, and related disorders that affect blood clotting.

Introduction to Coagulation Cascade

  • The coagulation cascade is a series of steps triggered by tissue injury to produce a stable blood clot.
  • Hemostasis is the process of stopping bleeding, with primary hemostasis forming a platelet plug and secondary hemostasis stabilizing it.

Pathways of the Coagulation Cascade

  • There are three interconnected pathways: extrinsic, intrinsic, and common.
  • The extrinsic pathway starts with tissue factor (factor III) exposure and activation of factor VII; it requires vitamin K.
  • The intrinsic pathway is triggered by factor XII contacting collagen and involves factors XII, XI, IX, and VIII.
  • Both pathways activate factor X, leading into the common pathway.

Common Pathway and Clot Formation

  • In the common pathway, factors Xa, Va, and calcium form a prothrombinase complex that converts prothrombin (II) to thrombin (IIa).
  • Thrombin converts fibrinogen (I) to fibrin (Ia) and activates factor XIII, which crosslinks fibrin to stabilize the clot.
  • Thrombin also helps activate platelets and factors V, VIII, and IX.

Fibrin Production and Clot Stabilization

  • Fibrin is produced at the cascade's end by thrombin-mediated cleavage of fibrinogen.
  • Fibrin forms a mesh that stabilizes the blood clot.

Coagulation Disorders

  • Coagulation disorders result from deficiencies in clotting factors, causing too much or too little clotting.
  • Von Willebrand disease (deficiency in von Willebrand factor) mainly affects primary hemostasis and stabilizes factor VIII.
  • Hemophilia A (factor VIII deficiency), B (factor IX deficiency), and C (factor XI deficiency) disrupt the intrinsic pathway.
  • Vitamin K deficiency impairs production of factors II, VII, IX, and X, affecting all pathways.

Key Terms & Definitions

  • Hemostasis — process to stop bleeding.
  • Clotting Factor — protein involved in forming a blood clot.
  • Zymogen — inactive form of an enzyme, such as clotting factors.
  • Thrombin (IIa) — enzyme that converts fibrinogen to fibrin.
  • Fibrin (Ia) — protein forming the meshwork of a blood clot.
  • Intrinsic Pathway — clotting triggered by factors inside blood.
  • Extrinsic Pathway — clotting triggered by tissue factor outside blood.
  • Common Pathway — final steps leading to clot formation.

Action Items / Next Steps

  • Review diagrams and sequential steps of the coagulation pathways.
  • Memorize the key clotting factors for each pathway and their order.
  • Understand the clinical relevance of major coagulation disorders.