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Which conditions are commonly treated with antacids?
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Peptic Ulcer Disease and Gastroesophageal Reflux Disease (GERD).
What forms are antacids available in, and what are the administration guidelines for each?
Chewable tablets (must be chewed thoroughly and followed by water or milk) and liquid form (should be shaken before taking).
How does aluminum hydroxide affect phosphate levels in the body?
It reduces phosphate absorption, which can lead to hypophosphatemia.
What are the primary purposes of antacids?
Neutralize gastric acid to reduce irritation and relieve heartburn symptoms, and enhance mucosal protection.
What should be done before taking a liquid antacid?
The liquid should be shaken before taking.
Why might magnesium hydroxide not be suitable for someone with renal impairment?
Because it can lead to magnesium accumulation in the body.
What are the potential gastrointestinal side effects of taking calcium carbonate as an antacid?
Constipation, belching, and flatulence.
When should antacids typically be taken?
1 to 3 hours after meals and at bedtime or when symptomatic.
Why should antacids be taken separately from other medications?
To prevent interference with the absorption and effectiveness of other medications.
Why is the timing of antacid administration important?
To maximize effectiveness and minimize interaction with the absorption of other medications.
Why should aluminum hydroxide antacids be used with caution in patients with hypertension or heart failure?
Because they have a high sodium content.
What effect does magnesium hydroxide have, and why might it be contraindicated?
It acts as a laxative and can cause diarrhea. It is contraindicated in intestinal obstruction and renal impairment due to the risk of magnesium accumulation.
What are common side effects of calcium carbonate antacids?
What is the potential impact of high sodium content in certain antacids?
It may exacerbate conditions like hypertension or heart failure.
What is a potential side effect of aluminum hydroxide in antacids?
Constipation and reduced phosphate absorption, leading to hypophosphatemia.
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